Targeted deletion of all isoforms of the trkC gene suggests the use of alternate receptors by its ligand neurotrophin-3 in neuronal development and implicates trkC in normal cardiogenesis

Ítems similars

• Evidence for a Role of Truncated trkC Receptor Isoforms in Mouse Development
  per: Palko, Mary Ellen, et al.
  Publicat: (1999)
• Absence of Sensory Neurons before Target Innervation in Brain-Derived Neurotrophic Factor-, Neurotrophin 3-, and TrkC-Deficient Embryonic Mice
  per: Liebl, Daniel J., et al.
  Publicat: (1997)
• The binding epitopes of neurotrophin-3 to its receptors trkC and gp75 and the design of a multifunctional human neurotrophin.
  per: Urfer, R, et al.
  Publicat: (1994)
• Diverse dependencies of developing Merkel innervation on the trkA and both full-length and truncated isoforms of trkC
  per: Cronk, Katharine M., et al.
  Publicat: (2002)
• A kinase-deficient TrkC receptor isoform activates Arf6–Rac1 signaling through the scaffold protein tamalin
  per: Esteban, Pedro F., et al.
  Publicat: (2006)