Hypothalamic Protein Kinase C Regulates Glucose Production

OBJECTIVE—A selective rise in hypothalamic lipid metabolism and the subsequent activation of SUR1/Kir6.2 ATP-sensitive K(+) (K(ATP)) channels inhibit hepatic glucose production. The mechanisms that link the ability of hypothalamic lipid metabolism to the activation of K(ATP) channels remain unknown....

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Detalhes bibliográficos
Main Authors: Ross, Rachel, Wang, Penny Y.T., Chari, Madhu, Lam, Carol K.L., Caspi, Liora, Ono, Hiraku, Muse, Evan D., Li, Xiaosong, Gutierrez-Juarez, Roger, Light, Peter E., Schwartz, Gary J., Rossetti, Luciano, Lam, Tony K.T.
Formato: Artigo
Idioma:Inglês
Publicado em: American Diabetes Association 2008
Assuntos:
Metabolism
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2494694/
https://ncbi.nlm.nih.gov/pubmed/18511848
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2337/db08-0206
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