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A pathogenic dyskerin mutation impairs proliferation and activates a DNA damage response independent of telomere length in mice

Telomeres are nucleoprotein structures that cap the ends of chromosomes, protecting them from exonucleases and distinguishing them from double-stranded breaks. Their integrity is maintained by telomerase, an enzyme consisting of a reverse transcriptase, TERT and an RNA template, TERC, and other comp...

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Bibliographische Detailangaben
Hauptverfasser: Gu, Bai-Wei, Bessler, Monica, Mason, Philip J.
Format: Artigo
Sprache:Inglês
Veröffentlicht: National Academy of Sciences 2008
Schlagworte:
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2481326/
https://ncbi.nlm.nih.gov/pubmed/18626023
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0803559105
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