Herpes simplex virus type 1 dUTPase mutants are attenuated for neurovirulence, neuroinvasiveness, and reactivation from latency.

Podobne zapisy

• Herpes simplex virus type 1 latency-associated transcription unit promotes anatomical site-dependent establishment and reactivation from latency.
  od: Sawtell, N M, i wsp.
  Wydane: (1992)
• Phosphorylation of Herpes Simplex Virus 1 dUTPase Upregulated Viral dUTPase Activity To Compensate for Low Cellular dUTPase Activity for Efficient Viral Replication
  od: Kato, Akihisa, i wsp.
  Wydane: (2014)
• The herpes simplex virus type 1 latency-associated transcript gene regulates the establishment of latency.
  od: Thompson, R L, i wsp.
  Wydane: (1997)
• Evidence that the Herpes Simplex Virus Type 1 ICP0 Protein Does Not Initiate Reactivation from Latency In Vivo
  od: Thompson, R. L., i wsp.
  Wydane: (2006)
• ICP34.5 mutants of herpes simplex virus type 1 strain 17syn+ are attenuated for neurovirulence in mice and for replication in confluent primary mouse embryo cell cultures.
  od: Bolovan, C A, i wsp.
  Wydane: (1994)