Binding sites for the herpes simplex virus immediate-early protein ICP4 impose an increased dependence on viral DNA replication on simple model promoters located in the viral genome.

Lignende værker

• Herpes Simplex Virus ICP0 and ICP34.5 Counteract Distinct Interferon-Induced Barriers to Virus Replication
  af: Mossman, Karen L., et al.
  Udgivet: (2002)
• Cells that constitutively express the herpes simplex virus immediate-early protein ICP4 allow efficient activation of viral delayed-early genes in trans.
  af: Persson, R H, et al.
  Udgivet: (1985)
• Herpes simplex virus immediate-early proteins ICP0 and ICP4 activate the endogenous human alpha-globin gene in nonerythroid cells.
  af: Cheung, P, et al.
  Udgivet: (1997)
• Herpes Simplex Virus 1 ICP0 Phosphorylation Site Mutants Are Attenuated for Viral Replication and Impaired for Explant-Induced Reactivation
  af: Mostafa, Heba H., et al.
  Udgivet: (2011)
• N-Terminal Phosphorylation Sites of Herpes Simplex Virus 1 ICP0 Differentially Regulate Its Activities and Enhance Viral Replication
  af: Mostafa, Heba H., et al.
  Udgivet: (2013)