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Defective γ-aminobutyric acid type B receptor-activated inwardly rectifying K(+ )currents in cerebellar granule cells isolated from weaver and Girk2 null mutant mice
Stimulation of inhibitory neurotransmitter receptors, such as γ-aminobutyric acid type B (GABA(B)) receptors, activates G protein-gated inwardly rectifying K(+) channels (GIRK) which, in turn, influence membrane excitability. Seizure activity has been reported in a Girk2 null mutant mouse lacking GI...
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| Hauptverfasser: | , , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
The National Academy of Sciences of the USA
1997
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC23753/ https://ncbi.nlm.nih.gov/pubmed/9342388 |
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