Intracerebral tumor-associated hemorrhage caused by overexpression of the vascular endothelial growth factor isoforms VEGF(121) and VEGF(165) but not VEGF(189)

The vascular endothelial growth factor (VEGF) has been shown to be a significant mediator of angiogenesis during a variety of normal and pathological processes, including tumor development. Human U87MG glioblastoma cells express the three VEGF isoforms: VEGF(121), VEGF(165), and VEGF(189). Here, we...

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Bibliographic Details
Main Authors: Cheng, Shi-Yuan, Nagane, Motoo, Huang, H.-J. Su, Cavenee, Webster K.
Format: Artigo
Language:Inglês
Published: National Academy of Sciences 1997
Subjects:
Biological Sciences
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC23709/
https://ncbi.nlm.nih.gov/pubmed/9342366
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