Abnormalities of pancreatic islets by targeted expression of a dominant-negative K(ATP) channel

ATP-sensitive K(+) (K(ATP)) channels are known to play important roles in various cellular functions, but the direct consequences of disruption of K(ATP) channel function are largely unknown. We have generated transgenic mice expressing a dominant-negative form of the K(ATP) channel subunit Kir6.2 (...

詳細記述

保存先:
書誌詳細
主要な著者: Miki, Takashi, Tashiro, Fumi, Iwanaga, Toshihiko, Nagashima, Kazuaki, Yoshitomi, Hideyuki, Aihara, Hiroyuki, Nitta, Yoshio, Gonoi, Tohru, Inagaki, Nobuya, Miyazaki, Jun-ichi, Seino, Susumu
フォーマット: Artigo
言語:Inglês
出版事項: The National Academy of Sciences of the USA 1997
主題:
Biological Sciences
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC23672/
https://ncbi.nlm.nih.gov/pubmed/9342346
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