A subset of SR proteins activates splicing of the cardiac troponin T alternative exon by direct interactions with an exonic enhancer.

Lignende værker

• Nucleotide substitutions within the cardiac troponin T alternative exon disrupt pre-mRNA alternative splicing.
  af: Cooper, T A, et al.
  Udgivet: (1989)
• Muscle-specific splicing enhancers regulate inclusion of the cardiac troponin T alternative exon in embryonic skeletal muscle.
  af: Ryan, K J, et al.
  Udgivet: (1996)
• The cardiac troponin T alternative exon contains a novel purine-rich positive splicing element.
  af: Xu, R, et al.
  Udgivet: (1993)
• Distinct functions of SR proteins in recruitment of U1 small nuclear ribonucleoprotein to alternative 5' splice sites.
  af: Zahler, A M, et al.
  Udgivet: (1995)
• Cis requirements for alternative splicing of the cardiac troponin T pre-mRNA.
  af: Cooper, T A, et al.
  Udgivet: (1988)