RPM2, independently of its mitochondrial RNase P function, suppresses an ISP42 mutant defective in mitochondrial import and is essential for normal growth.

RPM2 is identified here as a high-copy suppressor of isp42-3, a temperature-sensitive mutant allele of the mitochondrial protein import channel component, Isp42p. RPM2 already has an established role as a protein component of yeast mitochondrial RNase P, a ribonucleoprotein enzyme required for the 5...

Full description

Saved in:
Bibliographic Details
Main Authors: Kassenbrock, C K, Gao, G J, Groom, K R, Sulo, P, Douglas, M G, Martin, N C
Format: Artigo
Language:Inglês
Published: 1995
Subjects:
Research Article
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC230720/
https://ncbi.nlm.nih.gov/pubmed/7544434
Tags: Add Tag
No Tags, Be the first to tag this record!

Similar Items