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RPM2, independently of its mitochondrial RNase P function, suppresses an ISP42 mutant defective in mitochondrial import and is essential for normal growth.

RPM2 is identified here as a high-copy suppressor of isp42-3, a temperature-sensitive mutant allele of the mitochondrial protein import channel component, Isp42p. RPM2 already has an established role as a protein component of yeast mitochondrial RNase P, a ribonucleoprotein enzyme required for the 5...

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Detaylı Bibliyografya
Asıl Yazarlar: Kassenbrock, C K, Gao, G J, Groom, K R, Sulo, P, Douglas, M G, Martin, N C
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 1995
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC230720/
https://ncbi.nlm.nih.gov/pubmed/7544434
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