RPM2, independently of its mitochondrial RNase P function, suppresses an ISP42 mutant defective in mitochondrial import and is essential for normal growth.

RPM2 is identified here as a high-copy suppressor of isp42-3, a temperature-sensitive mutant allele of the mitochondrial protein import channel component, Isp42p. RPM2 already has an established role as a protein component of yeast mitochondrial RNase P, a ribonucleoprotein enzyme required for the 5...

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Εκδόθηκε σε:	Mol Cell Biol
Κύριοι συγγραφείς:	Kassenbrock, C K, Gao, G J, Groom, K R, Sulo, P, Douglas, M G, Martin, N C
Μορφή:	Artigo
Γλώσσα:	Inglês
Έκδοση:	Taylor & Francis 1995
Θέματα:	Research Article
Διαθέσιμο Online:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC230720/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7544434/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.15.9.4763
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