Dual functions of the AML1/Evi-1 chimeric protein in the mechanism of leukemogenesis in t(3;21) leukemias.

Benzer Materyaller

• Generation of the AML1-EVI-1 fusion gene in the t(3;21)(q26;q22) causes blastic crisis in chronic myelocytic leukemia.
  Yazar:: Mitani, K, ve diğerleri
  Baskı/Yayın Bilgisi: (1994)
• An acute myeloid leukemia gene, AML1, regulates hemopoietic myeloid cell differentiation and transcriptional activation antagonistically by two alternative spliced forms.
  Yazar:: Tanaka, T, ve diğerleri
  Baskı/Yayın Bilgisi: (1995)
• RUNX1-ETO and RUNX1-EVI1 Differentially Reprogram the Chromatin Landscape in t(8;21) and t(3;21) AML
  Yazar:: Loke, Justin, ve diğerleri
  Baskı/Yayın Bilgisi: (2017)
• The extracellular signal-regulated kinase pathway phosphorylates AML1, an acute myeloid leukemia gene product, and potentially regulates its transactivation ability.
  Yazar:: Tanaka, T, ve diğerleri
  Baskı/Yayın Bilgisi: (1996)
• The Evi-1 oncoprotein inhibits c-Jun N-terminal kinase and prevents stress-induced cell death
  Yazar:: Kurokawa, Mineo, ve diğerleri
  Baskı/Yayın Bilgisi: (2000)