CD59 or C3 are not requred for angiotensin II-dependent hypertension or hypertrophy in mice

Complement is a major pro-inflammatory innate immune system whose serum activity correlates with systolic blood pressure in humans. To date, no studies using in vivo models have directly examined the role of individual complement components in regulating vessel function, hypertension and cardiac hyp...

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Bibliographische Detailangaben
Hauptverfasser: Coles, Barbara, Lewis, Ruth, Anning, Peter B, Morton, Jonathan, Baalasubramanian, Sivasankar, Morgan, B Paul, O'Donnell, Valerie B
Format: Artigo
Sprache:Inglês
Veröffentlicht: Blackwell Science Inc 2007
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Original Articles
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2265976/
https://ncbi.nlm.nih.gov/pubmed/17433074
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1365-2567.2007.02598.x
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