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GPR30 Contributes to Estrogen-Induced Thymic Atrophy
The mechanisms by which prolonged estrogen exposures, such as estrogen therapy and pregnancy, reduce thymus weight, cellularity, and CD4 and CD8 phenotype expression, have not been well defined. In this study, the roles played by the membrane estrogen receptor, G protein-coupled receptor 30 (GPR30),...
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| Hauptverfasser: | , , , , , , , , , , , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
The Endocrine Society
2008
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2262170/ https://ncbi.nlm.nih.gov/pubmed/18063692 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1210/me.2007-0359 |
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