GPR30 Contributes to Estrogen-Induced Thymic Atrophy

The mechanisms by which prolonged estrogen exposures, such as estrogen therapy and pregnancy, reduce thymus weight, cellularity, and CD4 and CD8 phenotype expression, have not been well defined. In this study, the roles played by the membrane estrogen receptor, G protein-coupled receptor 30 (GPR30),...

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Detaylı Bibliyografya
Asıl Yazarlar: Wang, Chunhe, Dehghani, Babak, Magrisso, I. Jack, Rick, Elizabeth A., Bonhomme, Edna, Cody, David B., Elenich, Laura A., Subramanian, Sandhya, Murphy, Stephanie J., Kelly, Martin J., Rosenbaum, Jan S., Vandenbark, Arthur A., Offner, Halina
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: The Endocrine Society 2008
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2262170/
https://ncbi.nlm.nih.gov/pubmed/18063692
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1210/me.2007-0359
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