Potentiation of the Cardiac L-Type Ca(2+) Channel (α(1C)) by Dihydropyridine Agonist and Strong Depolarization Occur via Distinct Mechanisms

Eitemau Tebyg

• Tagging with green fluorescent protein reveals a distinct subcellular distribution of L-type and non-L-type Ca(2+) channels expressed in dysgenic myotubes
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• The α(1S) III-IV Loop Influences 1,4-Dihydropyridine Receptor Gating but Is Not Directly Involved in Excitation-Contraction Coupling Interactions with the Type 1 Ryanodine Receptor
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  Cyhoeddwyd: (2008)
• Ca(v)1.4α1 Subunits Can Form Slowly Inactivating Dihydropyridine-Sensitive L-Type Ca(2+) Channels Lacking Ca(2+)-Dependent Inactivation
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  Cyhoeddwyd: (2003)
• Fast activation of cardiac Ca++ channel gating charge by the dihydropyridine agonist, BAY K 8644.
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• Excitation–contraction coupling is unaffected by drastic alteration of the sequence surrounding residues L720–L764 of the α(1S) II-III loop
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  Cyhoeddwyd: (2001)