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ATP alters current fluctuations of cystic fibrosis transmembrane conductance regulator: evidence for a three-state activation mechanism

The cystic fibrosis gene product cystic fibrosis transmembrane conductance regulator (CFTR) is a low conductance, cAMP-regulated Cl- channel. Removal of cytosolic ATP causes a cessation of cAMP-dependent kinase-phosphorylated CFTR channel activity that resumes upon ATP addition. (Anderson, M. P., H....

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Natura: Artigo
Lingua:Inglês
Pubblicazione: The Rockefeller University Press 1994
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC2229193/
https://ncbi.nlm.nih.gov/pubmed/7525859
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