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On the Mechanism of MgATP-dependent Gating of CFTR Cl(−) Channels
CFTR, the product of the gene mutated in cystic fibrosis, is an ATPase that functions as a Cl(−) channel in which bursts of openings separate relatively long interburst closed times (τib). Channel gating is controlled by phosphorylation and MgATP, but the underlying molecular mechanisms remain contr...
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| Principais autores: | , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
The Rockefeller University Press
2003
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2217317/ https://ncbi.nlm.nih.gov/pubmed/12508051 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1085/jgp.20028673 |
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