Support of renal blood flow after ischaemic-reperfusion injury by endogenous formation of nitric oxide and of cyclo-oxygenase vasodilator metabolites.

1. Ischaemia-reperfusion injury in the kidney is associated with a loss of autoregulation, an increase in renal vascular resistance (RVR), a decrease of renal blood flow (RBF) and ultimately acute renal failure. The aim of this study was to investigate the role of the release of endogenous nitric ox...

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Bibliografiska uppgifter
Huvudupphovsmän: Cristol, J. P., Thiemermann, C., Mitchell, J. A., Walder, C., Vane, J. R.
Materialtyp: Artigo
Språk:Inglês
Publicerad: Nature Publishing Group 1993
Ämnen:
Research Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC2175578/
https://ncbi.nlm.nih.gov/pubmed/7684301
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