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The MEF2D transcription factor mediates stress-dependent cardiac remodeling in mice

The adult heart responds to excessive neurohumoral signaling and workload by a pathological growth response characterized by hypertrophy of cardiomyocytes and activation of a fetal program of cardiac gene expression. These responses culminate in diminished pump function, ventricular dilatation, wall...

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Detalhes bibliográficos
Main Authors: Kim, Yuri, Phan, Dillon, van Rooij, Eva, Wang, Da-Zhi, McAnally, John, Qi, Xiaoxia, Richardson, James A., Hill, Joseph A., Bassel-Duby, Rhonda, Olson, Eric N.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Clinical Investigation 2007
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2129240/
https://ncbi.nlm.nih.gov/pubmed/18079970
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI33255
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