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Single-channel study of the spasmodic mutation α1A52S in recombinant rat glycine receptors

Inherited defects in glycine receptors lead to hyperekplexia, or startle disease. A mutant mouse, spasmodic, that has a startle phenotype, has a point mutation (A52S) in the glycine receptor α1 subunit. This mutation reduces the sensitivity of the receptor to glycine, but the mechanism by which this...

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Bibliografiset tiedot
Päätekijät: Plested, Andrew J R, Groot-Kormelink, Paul J, Colquhoun, David, Sivilotti, Lucia G
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Blackwell Science Inc 2007
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC2075205/
https://ncbi.nlm.nih.gov/pubmed/17331994
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2006.126920
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