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Pluripotency deficit in clones overcome by clone–clone aggregation: epigenetic complementation?
Abnormal gene expression patterns in somatic cell clones and their attrition in utero are commonly considered a consequence of errors in nuclear reprogramming. We observe that mouse clone blastocysts have less than half the normal cell number, and that higher cell number correlates with correct expr...
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| Main Authors: | , , , , |
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| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
Oxford University Press
2003
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC204490/ https://ncbi.nlm.nih.gov/pubmed/14517267 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/cdg507 |
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