Continuous fat oxidation in acetyl–CoA carboxylase 2 knockout mice increases total energy expenditure, reduces fat mass, and improves insulin sensitivity

Acetyl–CoA carboxylase 2 (ACC)2 is a key regulator of mitochondrial fat oxidation. To examine the impact of ACC2 deletion on whole-body energy metabolism, we measured changes in substrate oxidation and total energy expenditure in Acc2(−/−) and WT control mice fed either regular or high-fat diets. To...

תיאור מלא

שמור ב:
מידע ביבליוגרפי
Main Authors: Choi, Cheol Soo, Savage, David B., Abu-Elheiga, Lutfi, Liu, Zhen-Xiang, Kim, Sheene, Kulkarni, Ameya, Distefano, Alberto, Hwang, Yu-Jin, Reznick, Richard M., Codella, Roberto, Zhang, Dongyan, Cline, Gary W., Wakil, Salih J., Shulman, Gerald I.
פורמט: Artigo
שפה:Inglês
יצא לאור: National Academy of Sciences 2007
נושאים:
Biological Sciences
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC2034222/
https://ncbi.nlm.nih.gov/pubmed/17923673
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0706794104
תגים: הוספת תג
אין תגיות, היה/י הראשונ/ה לתייג את הרשומה!

פריטים דומים