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Preferential block of inactivation-deficient Na(+) currents by capsaicin reveals a non-TRPV1 receptor within the Na(+) channel
Capsaicin elicits burning pain via the activation of the vanilloid receptor (TRPV1). Intriguingly, several reports showed that capsaicin also inhibits Na(+) currents but the mechanisms remain unclear. To explore this non-TRPV1 action we applied capsaicin to HEK293 cells stably expressing inactivatio...
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| Главные авторы: | , , |
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| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
2006
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1995448/ https://ncbi.nlm.nih.gov/pubmed/16962240 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.pain.2006.08.002 |
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