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The Kir6.2-F333I mutation differentially modulates K(ATP) channels composed of SUR1 or SUR2 subunits
Mutations in Kir6.2, the pore-forming subunit of the K(ATP) channel, that reduce the ability of ATP to block the channel cause neonatal diabetes. The stimulatory effect of MgATP mediated by the regulatory sulphonylurea receptor (SUR) subunit of the channel may also be modified. We compared the effec...
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| Главные авторы: | , |
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| Формат: | Artigo |
| Язык: | Inglês |
| Опубликовано: |
Blackwell Publishing Ltd
2007
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| Предметы: | |
| Online-ссылка: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1976406/ https://ncbi.nlm.nih.gov/pubmed/17395632 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/jphysiol.2007.130211 |
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