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JNK initiates a cytokine cascade that causes Pax2 expression and closure of the optic fissure

The c-Jun NH(2)-terminal kinase (JNK) group of mitogen-activated protein kinases is stimulated in response to a wide array of cellular stresses and proinflammatory cytokines. Mice lacking individual members of the Jnk family (Jnk1, Jnk2, and Jnk3) are viable and survive without overt structural abno...

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Dettagli Bibliografici
Pubblicato in:Genes Dev
Autori principali: Weston, Claire R., Wong, Anthony, Hall, J. Perry, Goad, Mary E.P., Flavell, Richard A., Davis, Roger J.
Natura: Artigo
Lingua:Inglês
Pubblicazione: Cold Spring Harbor Laboratory Press 2003
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Accesso online:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC196061/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12756228/
https://ncbi.nlm.nih.govhttps://doi.org/10.1101/gad.1087303
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