JNK initiates a cytokine cascade that causes Pax2 expression and closure of the optic fissure
The c-Jun NH(2)-terminal kinase (JNK) group of mitogen-activated protein kinases is stimulated in response to a wide array of cellular stresses and proinflammatory cytokines. Mice lacking individual members of the Jnk family (Jnk1, Jnk2, and Jnk3) are viable and survive without overt structural abno...
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| Pubblicato in: | Genes Dev |
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| Autori principali: | , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Cold Spring Harbor Laboratory Press
2003
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC196061/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12756228/ https://ncbi.nlm.nih.govhttps://doi.org/10.1101/gad.1087303 |
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