Chronic human immunodeficiency virus type 1 infection of myeloid cells disrupts the autoregulatory control of the NF-kappaB/Rel pathway via enhanced IkappaBalpha degradation.

Productive human immunodeficiency virus type 1 (HIV-1) infection causes sustained NF-kappaB DNA-binding activity in chronically infected monocytic cells. A direct temporal correlation exists between HIV infection and the appearance of NF-kappaB DNA-binding activity in myelomonoblastic PLB-985 cells....

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Veröffentlicht in:	J Virol
Hauptverfasser:	DeLuca, C, Roulston, A, Koromilas, A, Wainberg, M A, Hiscott, J
Format:	Artigo
Sprache:	Inglês
Veröffentlicht:	American Society for Microbiology (ASM) 1996
Schlagworte:	Research Article
Online-Zugang:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC190474/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8764027/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.70.8.5183-5193.1996
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