Apoptosis of T lymphocytes in experimental autoimmune encephalomyelitis. Evidence for programmed cell death as a mechanism to control inflammation in the brain.

Lignende værker

• Apoptotic Cell Death in Experimental Autoimmune Encephalomyelitis: Apoptosis of effector cells as a safe mechanism in the termination of an autoimmune inflammatory attack
  af: Chan, Andrew, et al.
  Udgivet: (2005)
• Functional and Pathogenic Differences of Th1 and Th17 Cells in Experimental Autoimmune Encephalomyelitis
  af: Domingues, Helena S., et al.
  Udgivet: (2010)
• Critical Role of the Programmed Death-1 (PD-1) Pathway in Regulation of Experimental Autoimmune Encephalomyelitis
  af: Salama, Alan D., et al.
  Udgivet: (2003)
• Role of passive T-cell death in chronic experimental autoimmune encephalomyelitis
  af: Issazadeh, Shohreh, et al.
  Udgivet: (2000)
• Oestrogen modulates experimental autoimmune encephalomyelitis and interleukin-17 production via programmed death 1
  af: Wang, Chunhe, et al.
  Udgivet: (2009)