A novel translational regulation function for the simian virus 40 large-T antigen gene.
Cells use the interferon-induced, double-stranded-RNA-dependent protein kinase PKR as a defense against virus infections. Upon activation, PKR phosphorylates and thereby inactivates the protein synthesis initiation factor eIF-2, resulting in the cessation of protein synthesis. Viruses have evolved v...
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| 出版年: | J Virol |
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| 主要な著者: | , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Microbiology (ASM)
1995
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC188643/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7815544/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/jvi.69.2.785-795.1995 |
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