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Expression of Nuclear β-Catenin and c-myc Is Correlated with Tumor Size but Not with Proliferative Activity of Colorectal Adenomas

Most colorectal cancers have loss-of-function mutations in the adenomatosis polyposis coli (APC) tumor suppressor gene. This leads to the accumulation of nuclear β-catenin, which, together with the DNA-binding protein TCF-4, functions as a transcriptional activator. The recently defined target genes...

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Main Authors: Brabletz, Thomas, Herrmann, Kathrin, Jung, Andreas, Faller, Gerhard, Kirchner, Thomas
Formato: Artigo
Idioma:Inglês
Publicado: American Society for Investigative Pathology 2000
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Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC1876834/
https://ncbi.nlm.nih.gov/pubmed/10702403
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