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Mice Lacking Smad3 Are Protected Against Cutaneous Injury Induced by Ionizing Radiation
Transforming growth factor-β (TGF-β) plays a central role in the pathogenesis of inflammatory and fibrotic diseases, including radiation-induced fibrosis. We previously reported that mice null for Smad3, a key downstream mediator of TGF-β, show accelerated healing of cutaneous incisional wounds with...
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| 主要な著者: | , , , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Investigative Pathology
2002
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1867194/ https://ncbi.nlm.nih.gov/pubmed/11891202 |
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