Full-length amyloid-beta (1-42(43)) and amino-terminally modified and truncated amyloid-beta 42(43) deposit in diffuse plaques.

Podobne zapisy

• Predominant deposition of amyloid-beta 42(43) in plaques in cases of Alzheimer's disease and hereditary cerebral hemorrhage associated with mutations in the amyloid precursor protein gene.
  od: Mann, D. M., i wsp.
  Wydane: (1996)
• Amyloid beta protein deposition in normal aging has the same characteristics as that in Alzheimer's disease. Predominance of A beta 42(43) and association of A beta 40 with cored plaques.
  od: Fukumoto, H., i wsp.
  Wydane: (1996)
• Distinct spatiotemporal accumulation of N-truncated and full-length amyloid-β(42) in Alzheimer’s disease
  od: Shinohara, Mitsuru, i wsp.
  Wydane: (2017)
• Amyloid beta 1-42 deposits do not lead to Alzheimer's neuritic plaques in aged dogs.
  od: Wisniewski, T, i wsp.
  Wydane: (1996)
• Amyloid-beta peptide Aβp3-42 affects early aggregation of full-length Aβ1-42
  od: Sanders, Hiromi M., i wsp.
  Wydane: (2009)