Protection Against Endotoxic Shock as a Consequence of Reduced Nitrosative Stress in MLCK210-Null Mice

This study investigated the consequences of deletion of the long isoform of myosin light chain kinase (MLCK210) on the cardiovascular changes induced by the bacterial endotoxin lipopolysaccharide (LPS) and cecal ligation puncture using MLCK210(−/−) mice. Here, we provide evidence that deletion of ML...

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Bibliografiske detaljer
Main Authors: Ranaivo, Hantamalala Ralay, Carusio, Nunzia, Wangensteen, Rosemary, Ohlmann, Patrick, Loichot, Cecile, Tesse, Angela, Chalupsky, Karel, Lobysheva, Irina, Haiech, Jacques, Watterson, D. Martin, Andriantsitohaina, Ramaroson
Format: Artigo
Sprog:Inglês
Udgivet: American Society for Investigative Pathology 2007
Fag:
Regular Articles
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC1851870/
https://ncbi.nlm.nih.gov/pubmed/17255312
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2353/ajpath.2007.060219
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