Protection Against Endotoxic Shock as a Consequence of Reduced Nitrosative Stress in MLCK210-Null Mice

This study investigated the consequences of deletion of the long isoform of myosin light chain kinase (MLCK210) on the cardiovascular changes induced by the bacterial endotoxin lipopolysaccharide (LPS) and cecal ligation puncture using MLCK210(−/−) mice. Here, we provide evidence that deletion of ML...

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Главные авторы: Ranaivo, Hantamalala Ralay, Carusio, Nunzia, Wangensteen, Rosemary, Ohlmann, Patrick, Loichot, Cecile, Tesse, Angela, Chalupsky, Karel, Lobysheva, Irina, Haiech, Jacques, Watterson, D. Martin, Andriantsitohaina, Ramaroson
Формат: Artigo
Язык:Inglês
Опубликовано: American Society for Investigative Pathology 2007
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Regular Articles
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC1851870/
https://ncbi.nlm.nih.gov/pubmed/17255312
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2353/ajpath.2007.060219
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