Protection Against Endotoxic Shock as a Consequence of Reduced Nitrosative Stress in MLCK210-Null Mice

This study investigated the consequences of deletion of the long isoform of myosin light chain kinase (MLCK210) on the cardiovascular changes induced by the bacterial endotoxin lipopolysaccharide (LPS) and cecal ligation puncture using MLCK210(−/−) mice. Here, we provide evidence that deletion of ML...

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Main Authors: Ranaivo, Hantamalala Ralay, Carusio, Nunzia, Wangensteen, Rosemary, Ohlmann, Patrick, Loichot, Cecile, Tesse, Angela, Chalupsky, Karel, Lobysheva, Irina, Haiech, Jacques, Watterson, D. Martin, Andriantsitohaina, Ramaroson
Formáid: Artigo
Teanga:Inglês
Foilsithe: American Society for Investigative Pathology 2007
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Regular Articles
Rochtain Ar Líne:https://ncbi.nlm.nih.gov/pmc/articles/PMC1851870/
https://ncbi.nlm.nih.gov/pubmed/17255312
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.2353/ajpath.2007.060219
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