Nitric oxide protects cultured rheumatoid synovial cells from Fas-induced apoptosis by inhibiting caspase-3

Nitric oxide (NO) is elevated in the synovial fluids and sera of patients with rheumatoid arthritis (RA) and is thought to be an important proinflammatory mediator in the rheumatoid synovium. To test the hypothesis that NO might modulate the apoptosis-inducing signal pathway, we investigated the eff...

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Главные авторы: Migita, Kiyoshi, Yamasaki, Satoshi, Kita, Masako, Ida, Hiroaki, Shibatomi, Kazutaka, Kawakami, Atsushi, Aoyagi, Takahiko, Eguchi, Katsuni
Формат: Artigo
Язык:Inglês
Опубликовано: Blackwell Science Inc 2001
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Original Articles
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC1783248/
https://ncbi.nlm.nih.gov/pubmed/11454065
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1046/j.1365-2567.2001.01252.x
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