Tumor necrosis factor alpha and interleukin-12 contribute to resistance to the intracellular bacterium Brucella abortus by different mechanisms.
Both interleukin-12 (IL-12) and tumor necrosis factor alpha (TNF-alpha) are produced early in intracellular bacterial infection. Depletion of either IL-12 or TNF-alpha by a single injection of specific antibody 4 h before the injection of Brucella abortus 19 led to the exacerbation of infection 2 we...
שמור ב:
| הוצא לאור ב: | Infect Immun |
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| Principais autores: | , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Society for Microbiology (ASM)
1996
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC174139/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/8698508/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/iai.64.7.2782-2786.1996 |
| תגים: |
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