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RecFOR proteins are essential for Pol V-mediated translesion synthesis and mutagenesis
When the replication fork moves through the template DNA containing lesions, daughter-strand gaps are formed opposite lesion sites. These gaps are subsequently filled-in either by translesion synthesis (TLS) or by homologous recombination. RecA filaments formed within these gaps are key intermediate...
Tallennettuna:
| Päätekijät: | , , |
|---|---|
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
Nature Publishing Group
2006
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1698908/ https://ncbi.nlm.nih.gov/pubmed/17139245 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.emboj.7601474 |
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