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RecFOR proteins are essential for Pol V-mediated translesion synthesis and mutagenesis

When the replication fork moves through the template DNA containing lesions, daughter-strand gaps are formed opposite lesion sites. These gaps are subsequently filled-in either by translesion synthesis (TLS) or by homologous recombination. RecA filaments formed within these gaps are key intermediate...

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Hlavní autoři: Fujii, Shingo, Isogawa, Asako, Fuchs, Robert P
Médium: Artigo
Jazyk:Inglês
Vydáno: Nature Publishing Group 2006
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC1698908/
https://ncbi.nlm.nih.gov/pubmed/17139245
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.emboj.7601474
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