JNK phosphorylation relieves HDAC3-dependent suppression of the transcriptional activity of c-Jun

The AP-1 transcription factor c-Jun is a prototypical nuclear effector of the JNK signal transduction pathway. The integrity of JNK phosphorylation sites at serines 63/73 and at threonines 91/93 in c-Jun is essential for signal-dependent target gene activation. We show that c-Jun phosphorylation med...

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Detalles Bibliográficos

Publicado en:	EMBO J
Principais autores:	Weiss, Carsten, Schneider, Sandra, Wagner, Erwin F., Zhang, Xiaohong, Seto, Edward, Bohmann, Dirk
Formato:	Artigo
Idioma:	Inglês
Publicado:	Nature Publishing Group 2003
Assuntos:	Articles
Acceso en liña:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC165634/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12853483/ https://ncbi.nlm.nih.govhttps://doi.org/10.1093/emboj/cdg364
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