JNK phosphorylation relieves HDAC3-dependent suppression of the transcriptional activity of c-Jun
The AP-1 transcription factor c-Jun is a prototypical nuclear effector of the JNK signal transduction pathway. The integrity of JNK phosphorylation sites at serines 63/73 and at threonines 91/93 in c-Jun is essential for signal-dependent target gene activation. We show that c-Jun phosphorylation med...
Kaydedildi:
| Yayımlandı: | EMBO J |
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| Asıl Yazarlar: | , , , , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
Nature Publishing Group
2003
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC165634/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12853483/ https://ncbi.nlm.nih.govhttps://doi.org/10.1093/emboj/cdg364 |
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