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JNK phosphorylation relieves HDAC3-dependent suppression of the transcriptional activity of c-Jun

The AP-1 transcription factor c-Jun is a prototypical nuclear effector of the JNK signal transduction pathway. The integrity of JNK phosphorylation sites at serines 63/73 and at threonines 91/93 in c-Jun is essential for signal-dependent target gene activation. We show that c-Jun phosphorylation med...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: Weiss, Carsten, Schneider, Sandra, Wagner, Erwin F., Zhang, Xiaohong, Seto, Edward, Bohmann, Dirk
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: Oxford University Press 2003
Gaiak:
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC165634/
https://ncbi.nlm.nih.gov/pubmed/12853483
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/cdg364
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