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JNK phosphorylation relieves HDAC3-dependent suppression of the transcriptional activity of c-Jun
The AP-1 transcription factor c-Jun is a prototypical nuclear effector of the JNK signal transduction pathway. The integrity of JNK phosphorylation sites at serines 63/73 and at threonines 91/93 in c-Jun is essential for signal-dependent target gene activation. We show that c-Jun phosphorylation med...
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| Autori principali: | , , , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
Oxford University Press
2003
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC165634/ https://ncbi.nlm.nih.gov/pubmed/12853483 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/emboj/cdg364 |
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