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Life with a Single Isoform of Akt: Mice Lacking Akt2 and Akt3 Are Viable but Display Impaired Glucose Homeostasis and Growth Deficiencies

To address the issues of isoform redundancy and isoform specificity of the Akt family of protein kinases in vivo, we generated mice deficient in both Akt2 and Akt3. In these mice, only the Akt1 isoform remains to perform essential Akt functions, such as glucose homeostasis, proliferation, differenti...

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Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: Dummler, Bettina , Tschopp, Oliver, Hynx, Debby, Yang, Zhong-Zhou, Dirnhofer, Stephan, Hemmings, Brian A.
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: American Society for Microbiology 2006
Gaiak:
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC1636753/
https://ncbi.nlm.nih.gov/pubmed/16923958
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/MCB.00722-06
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