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Exit from Arsenite-Induced Mitotic Arrest Is p53 Dependent
BACKGROUND: Arsenic is both a human carcinogen and a chemotherapeutic agent, but the mechanism of neither arsenic-induced carcinogenesis nor tumor selective cytotoxicity is clear. Using a model cell line in which p53 expression is regulated exogenously in a tetracycline-off system (TR9-7 cells), our...
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| Autors principals: | , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
National Institute of Environmental Health Sciences
2006
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1570045/ https://ncbi.nlm.nih.gov/pubmed/16966095 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1289/ehp.8969 |
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