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Deconstructing endothelial dysfunction: soluble guanylyl cyclase oxidation and the NO resistance syndrome
In this issue of the JCI, Stasch and colleagues suggest that a novel drug, BAY 58-2667, potently activates a pool of oxidized and heme-free soluble guanylyl cyclase (sGC; see the related article beginning on page 2552). The increased vasodilatory potency of BAY 58-2667 the authors found in a number...
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Clinical Investigation
2006
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| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1555666/ https://ncbi.nlm.nih.gov/pubmed/16955136 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI29807 |
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