GLUT4, AMP kinase, but not the insulin receptor, are required for hepatoportal glucose sensor–stimulated muscle glucose utilization

Lignende værker

• Normal hepatic glucose production in the absence of GLUT2 reveals an alternative pathway for glucose release from hepatocytes
  af: Guillam, Marie-Thérèse, et al.
  Udgivet: (1998)
• Normal kinetics of intestinal glucose absorption in the absence of GLUT2: Evidence for a transport pathway requiring glucose phosphorylation and transfer into the endoplasmic reticulum
  af: Stümpel, Frank, et al.
  Udgivet: (2001)
• Regulation of glucagon secretion by glucose transporter type 2 (glut2) and astrocyte-dependent glucose sensors
  af: Marty, Nell, et al.
  Udgivet: (2005)
• Regulation of Insulin-Stimulated Glucose Transporter GLUT4 Translocation and Akt Kinase Activity by Ceramide
  af: Summers, Scott A., et al.
  Udgivet: (1998)
• GLUT4 facilitates insulin stimulation and cAMP-mediated inhibition of glucose transport.
  af: Lawrence, J C, et al.
  Udgivet: (1992)