Hypoxia-induced endocytosis of Na,K-ATPase in alveolar epithelial cells is mediated by mitochondrial reactive oxygen species and PKC-ζ

Eitemau Tebyg

• α1-AMP-Activated Protein Kinase Regulates Hypoxia-Induced Na,K-ATPase Endocytosis via Direct Phosphorylation of Protein Kinase Cζ
  gan: Gusarova, Galina A., et al.
  Cyhoeddwyd: (2009)
• Phosphorylation of Adaptor Protein–2 μ2 Is Essential for Na(+),K(+)-ATPase Endocytosis in Response to Either G Protein–Coupled Receptor or Reactive Oxygen Species
  gan: Chen, Zongpei, et al.
  Cyhoeddwyd: (2006)
• Phosphorylation and ubiquitination are necessary for Na,K-ATPase endocytosis during hypoxia
  gan: Dada, Laura A., et al.
  Cyhoeddwyd: (2007)
• Mitochondrial reactive oxygen species are required for hypoxia-induced degradation of keratin intermediate filaments
  gan: Na, Ni, et al.
  Cyhoeddwyd: (2010)
• AMP-activated protein kinase regulates CO(2)-induced alveolar epithelial dysfunction in rats and human cells by promoting Na,K-ATPase endocytosis
  gan: Vadász, István, et al.
  Cyhoeddwyd: (2008)