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Paradoxical resistance to diet-induced obesity in UCP1-deficient mice

The availability of mice lacking the mitochondrial uncoupling protein UCP1, has provided an opportunity to analyze the relationship between the capacity for energy expenditure and the development of obesity in response to a high-fat, high-sucrose diet. Congenic UCP1-deficient mice on a C57BL/6J gene...

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Autores principales: Liu, Xiaotuan, Rossmeisl, Martin, McClaine, Jennifer, Kozak, Leslie P.
Formato: Artigo
Lenguaje:Inglês
Publicado: American Society for Clinical Investigation 2003
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Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC151850/
https://ncbi.nlm.nih.gov/pubmed/12569166
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI200315737
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