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Paradoxical resistance to diet-induced obesity in UCP1-deficient mice

The availability of mice lacking the mitochondrial uncoupling protein UCP1, has provided an opportunity to analyze the relationship between the capacity for energy expenditure and the development of obesity in response to a high-fat, high-sucrose diet. Congenic UCP1-deficient mice on a C57BL/6J gene...

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Detalhes bibliográficos
Main Authors: Liu, Xiaotuan, Rossmeisl, Martin, McClaine, Jennifer, Kozak, Leslie P.
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Clinical Investigation 2003
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC151850/
https://ncbi.nlm.nih.gov/pubmed/12569166
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI200315737
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