Overexpression of endothelial nitric oxide synthase accelerates atherosclerotic lesion formation in apoE-deficient mice

Nitric oxide (NO) derived from endothelial NO synthase (eNOS) is regarded as a protective factor against atherosclerosis. Therefore, augmentation of eNOS expression or NO production by pharmacological intervention is postulated to inhibit atherosclerosis. We crossed eNOS-overexpressing (eNOS-Tg) mic...

Täydet tiedot

Bibliografiset tiedot

Julkaisussa:	J Clin Invest
Päätekijät:	Ozaki, Masanori, Kawashima, Seinosuke, Yamashita, Tomoya, Hirase, Tetsuaki, Namiki, Masayuki, Inoue, Nobutaka, Hirata, Ken-ichi, Yasui, Hiroyuki, Sakurai, Hiromu, Yoshida, Yuichi, Masada, Masahiro, Yokoyama, Mitsuhiro
Aineistotyyppi:	Artigo
Kieli:	Inglês
Julkaistu:	American Society for Clinical Investigation 2002
Aiheet:	Article
Linkit:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC151086/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12163452/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI15215
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