Overexpression of Endothelial Nitric Oxide Synthase in Endothelial Cells Is Protective against Ischemia-Reperfusion Injury in Mouse Skeletal Muscle

Microvascular injury has been proposed to be a main cause of ischemia-reperfusion (I/R) injury. The roles of endothelial nitric oxide synthase (eNOS)-derived NO, a key regulator of vascular function, in I/R injury are incompletely understood. We used transgenic mice overexpressing eNOS in endothelia...

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Main Authors: Ozaki, Masanori, Kawashima, Seinosuke, Hirase, Tetsuaki, Yamashita, Tomoya, Namiki, Masayuki, Inoue, Nobutaka, Hirata, Ken-ichi, Yokoyama, Mitsuhiro
Formato: Artigo
Idioma:Inglês
Publicado: American Society for Investigative Pathology 2002
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Regular Articles
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC1867198/
https://ncbi.nlm.nih.gov/pubmed/11943718
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